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Thank you. That makes sense. It also ties in with the explanation given by my Dr. at Oregon Health Science University (OHSU). She explained why MDS/AML is so much more difficult than chronic myeloid leukemia (CML). There are many more variations in chromosome structures. Dr. Drucker at OHSU helped prove it was possible to shut down the mutations driving (CML) cancer without harming healthy cells.
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age 70, dx RAEB-2 on 11-26-2013 w/11% blasts. 8 cycles Vidaza 3w/Revlimid. SCT 8/15/2014, relapsed@Day+210 (AML). Now(SCT-Day+1005). Prepping w/ 10 days Dacogen for DLI on 6/9/2017.
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