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That sounds counter-intuitive to me. So please elaborate. My impression is that the "lack" of O2 is what stimulates the kidneys to release EPO. Is the premise that your body likes the O2 boost and when you withdraw it, the perceived lack of O2(even though it's normal) tricks the kidneys?
Unfortunately, for most with SAA, there's no shortage of EPO. Most have unusually high EPO levels.
Thanks for posting this. It pretty amazing and you are right, no one is going to try to mainstream something like this as long as profits procrit is bringing the $$$$s.
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Marlene, wife to John DX w/SAA April 2002, Stable partial remission; Treated with High Dose Cytoxan, Johns Hopkins, June 2002. Final phlebotomy 11/2016. As of July 2021 HGB 12.0, WBC 4.70/ANC 3.85, Plts 110K.
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